Labels:text | screenshot | black | black and white | font OCR: tic and The mechanism behind the antinatriun the parallelother renal effects of GH is not clear, but ase in time course of renal effects, and the incr or (IGF-I), levels of plasma insulin-like growth fac addition, suggest that it may be IGF-I mediated. In significant activation of the llowing renin-angiotensin-aldosterone system fc ig. 2.32). GH treatment has been found to occur (
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